Header graphic for print

Food Safety News

Breaking news for everyone's consumption

Grand Jury Indicts Meat Company Owners in Nebraska

A federal grand jury in Nebraska has indicted Paul Rosberg, 61, and Kelly Rosberg, 44, on six counts stemming from the sale of misbranded and/or non-inspected meat and meat products to Omaha Public Schools. The couple are from Wausa, NE.

If convicted, each could be sentenced to 3 to 5 years in federal prison, and fined from $10,000 to $250,000 on each of the six counts along with requirements that any release be supervised and fees paid.

Count one charges the pair with conspiracy with the intent to defraud. The second count charges Kelly Rosberg with selling 2,600 pounds of ground beef that was labeled as inspected by the USDA when it was not.

Count three charges both of the accused with selling on or about Sept. 19, 2011 ground beef that was not inspected by USDA. In other words, the indictment is for both the mislabeling ground beef as inspected when it was not and for actually selling beef outside the required USDA inspection.

The fourth count against both men is for representing the 2,600 pounds of beet was USDA inspected, Counts five and six are against Paul Rosberg for making a false statements on or about Nov. 3, 2011 to USDA’s Food Safety and Inspection Service.

Deborah Gilg, U.S. District Attorney for Nebraska, announced the indictments.

The pair were caught by a joint investigation of USDA’s Office of Program Evaluation, Enforcement and Review (OPEER) and the Inspector General (IG).

Information development by investigators led to the issuance of a search warrant for Nebraska’s Finest Meats, which led to the confiscation of records, labels, equipment and other evidence in the case.

Nebraska’s Finest Meats has suspended operations.

Omaha Public Schools, with about 50,000 K-12 students, are Nebraska’s largest.

© Food Safety News
  • http://downercattle.blogspot.com/2012/09/sale-of-misbranded-andor-non-inspected.html Terry S. Singeltary Sr.

    Great news that the federal grand jury in Nebraska indicted these folks that might have poisoned our children.
    Good job USDA et al, after the fact, that our children, once again, were exposed to misbranded and/or non-inspected meat and meat products, which could include mad cow type disease i.e. typical or atypical BSE, and or other deadly pathogens, but you got your man.
    but, who will watch the children now ?
    WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE NEXT 50 YEARS ?
    > > > Ackerman says downed cattle are 50 times more likely to have mad cow disease (also known as Bovine Spongiform Encephalopathy, or BSE) than ambulatory cattle that are suspected of having BSE. Of the 20 confirmed cases of mad cow disease in North America since 1993, at least 16 have involved downer cattle, he said. < < <
    http://ackerman.house.gov/index.cfm?sectionid=318&itemid=1828
    WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE NEXT 50 YEARS ???
    don’t kid yourself like the USDA et al do about sporadic CJD. USDA will not tell the mothers this ;
    Thursday, August 12, 2010
    Seven main threats for the future linked to prions
    First threat
    The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed. ***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.
    Second threat
    snip…
    http://www.neuroprion.org/en/np-neuroprion.html
    EFSA Journal 2011 The European Response to BSE: A Success Story
    This is an interesting editorial about the Mad Cow Disease debacle, and it’s ramifications that will continue to play out for decades to come ;
    Monday, October 10, 2011
    EFSA Journal 2011 The European Response to BSE: A Success Story
    snip…
    EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far but the possibility that a small proportion of human cases so far classified as “sporadic” CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.
    snip…
    http://www.efsa.europa.eu/en/efsajournal/pub/e991.htm?emt=1
    http://www.efsa.europa.eu/en/efsajournal/doc/e991.pdf
    see follow-up here about North America BSE Mad Cow TSE prion risk factors, and the ever emerging strains of Transmissible Spongiform Encephalopathy in many species here in the USA, including humans ;
    http://transmissiblespongiformencephalopathy.blogspot.com/2011/10/efsa-journal-2011-european-response-to.html
    2010-2011
    When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.
    This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle.
    http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2
    Saturday, May 26, 2012
    Are USDA assurances on mad cow case ‘gross oversimplification’?
    SNIP…
    What irks many scientists is the USDA’s April 25 statement that the rare disease is “not generally associated with an animal consuming infected feed.”
    The USDA’s conclusion is a “gross oversimplification,” said Dr. Paul Brown, one of the world’s experts on this type of disease who retired recently from the National Institutes of Health. “(The agency) has no foundation on which to base that statement.”
    “We can’t say it’s not feed related,” agreed Dr. Linda Detwiler, an official with the USDA during the Clinton Administration now at Mississippi State.
    In the May 1 email to me, USDA’s Cole backed off a bit. “No one knows the origins of atypical cases of BSE,” she said
    The argument about feed is critical because if feed is the cause, not a spontaneous mutation, the California cow could be part of a larger outbreak.
    SNIP…
    http://bseusa.blogspot.com/2012/05/are-usda-assurances-on-mad-cow-case.html
    ==============================================
    Saturday, August 4, 2012
    Final Feed Investigation Summary – California BSE Case – July 2012
    http://transmissiblespongiformencephalopathy.blogspot.com/2012/08/final-feed-investigation-summary.html
    =============================================
    SUMMARY REPORT CALIFORNIA BOVINE SPONGIFORM ENCEPHALOPATHY CASE INVESTIGATION JULY 2012
    Summary Report BSE 2012
    Executive Summary
    http://transmissiblespongiformencephalopathy.blogspot.com/2012/08/summary-report-california-bovine.html
    Saturday, August 4, 2012
    Update from APHIS Regarding Release of the Final Report on the BSE Epidemiological Investigation
    http://transmissiblespongiformencephalopathy.blogspot.com/2012/08/update-from-aphis-regarding-release-of.html
    WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE NEXT 50 YEARS ???
    Saturday, May 2, 2009
    U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK DOWNER COW SCHOOL LUNCH PROGRAM
    http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html
    OUR SCHOOL CHILDREN ALL ACROSS THE USA WERE FED THE MOST HIGH RISK CATTLE FOR MAD COW DISEASE FOR 4 YEARS I.E. DEAD STOCK DOWNER CATTLE VIA THE USDA AND THE NSLP.
    WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5+ DECADES ???
    DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ???
    you can check and see here ;
    http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf
    http://downercattle.blogspot.com/
    the article in question was an opinion article _written_ by Dr. Richard Raymond former Undersecretary for Food Safety, U.S. Department of Agriculture (2005-2008), and published on Bill Marlers Food Safety News feed.
    http://www.foodsafetynews.com/2012/08/central-valley-meat-company-usda-did-its-job-ok/?utm_source=newsletter&utm_medium=email&utm_campaign=120827
    http://www.foodsafetynews.com/
    http://www.foodsafetynews.com/about-us/
    Sunday, August 26, 2012
    Detection of PrPSc in peripheral tissues of clinically affected cattle after oral challenge with BSE
    http://transmissiblespongiformencephalopathy.blogspot.com/2012/08/detection-of-prpsc-in-peripheral.html
    in the url that follows, I have posted
    SRM breaches first, as late as 2011.
    then
    MAD COW FEED BAN BREACHES AND TONNAGES OF MAD COW FEED IN COMMERCE up until 2007, when they ceased posting them.
    then,
    MAD COW SURVEILLANCE BREACHES.
    Friday, May 18, 2012
    Update from APHIS Regarding a Detection of Bovine Spongiform Encephalopathy (BSE) in the United States Friday May 18, 2012
    http://transmissiblespongiformencephalopathy.blogspot.com/2012/05/update-from-aphis-regarding-detection.html
    2011 Monday, September 26, 2011
    L-BSE BASE prion and atypical sporadic CJD
    http://bse-atypical.blogspot.com/2011/09/l-bse-base-prion-and-atypical-sporadic.html
    Tuesday, June 26, 2012
    Creutzfeldt Jakob Disease Human TSE report update North America, Canada, Mexico, and USDA PRION UNIT as of May 18, 2012
    type determination pending Creutzfeldt Jakob Disease (tdpCJD), is on the rise in Canada and the USA
    http://creutzfeldt-jakob-disease.blogspot.com/2012/06/creutzfeldt-jakob-disease-human-tse.html
    Monday, July 23, 2012
    The National Prion Disease Pathology Surveillance Center July 2012
    http://prionunitusaupdate2008.blogspot.com/2012/07/the-national-prion-disease-pathology.html
    after-the-fact litigation, on a slow incubating disease, one that is 100% fatal, once clinical, is like robbing the dead, and their families.
    this is why corporate America will not let science investigate sporadic CJD strains $$$
    what will history show us from a deja vu incident like what happened in the UK with their mad cow debacle?
    as the decades pass, will our children, and adults, be as lucky as the UK victims of corporate greed were, with their genetic susceptibility and exposer to their strain of BSE aka mad cow disease ?
    OR, will North America population be more susceptible to the ever growing list of different strains of different TSE prion disease (aka mad cow type disease), typical and atypical strains growing, in many different species, all of which have been fed back to food producing animals and humans in North America, including our children, thanks to the continued disregard, and after-the-fact findings, by the USDA et al ?
    tune in 10, 20, –, 50 years from now, if were still here. …end…tss
    3. I asked the slaughterhouses workers in detail to explain the routine slaughtering practices and was astonished to learn that sheep’s brains were generally left inside the skull whereas cattle brains were routinely removed to be added to our “meat products” – meat pies, pates, tinned items and stock cubes. I therefore feared that this almost indestructible infective agent was being swallowed by all beef eaters in the UK in large doses which would inevitably infect genetically susceptible people.
    http://bseinquiry.blogspot.com/2012/04/wendy-grant-who-has-died-aged-89-was.html
    Sunday, May 18, 2008
    BSE, CJD, and Baby foods (the great debate 1999 to 2005)
    http://bseinquiry.blogspot.com/2008/05/bse-cjd-and-baby-foods-great-debate.html
    deja vu
    Wednesday, March 14, 2012
    PINK SLIME, MRM’s, BSE AKA MAD COW DISEASE, AND THE USDA NSLP
    http://madcowusda.blogspot.com/2012/03/pink-slime-mrms-bse-aka-mad-cow-disease.html
    layperson
    Terry S. Singeltary SR. P.O. Box 42 Bacliff, Texas USA 77518 flounder9@verizon.net
    Monday, September 3, 2012
    Sale of misbranded and/or non-inspected meat and meat products to Omaha Public Schools indicted
    http://downercattle.blogspot.com/2012/09/sale-of-misbranded-andor-non-inspected.html