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Foreign Markets Cautious on Canadian Beef Pending BSE Investigation

Foreign markets are being extra cautious about Canadian beef until an investigation into an Alberta cow found to be infected with bovine spongiform encephalopathy (BSE) — or, as it is commonly called, “mad cow disease” — runs its course.

In the meantime, South Korea has temporarily banned imports of Canadian beef as a precaution.

None of the BSE-infected beef was allowed to reach human or animal feed markets, according to Canadian officials. Meanwhile, the northern Alberta farm from which the infected sample came is reportedly under quarantine.

Confirmed on Feb. 11 by the Canadian Food Inspection Agency, the Alberta cow is the first in Canada with BSE since 2011. BSE is a progressive, fatal neurological disease found in cattle. It is believed to be caused by prion-contaminated feed.

The next steps in the investigation are additional laboratory tests and collection of further information on the animal, its offspring, and its herd of origin to determine how and when it contracted the disease. CFIA’s investigation will focus on the feed supplied to the infected cow during its first year.

Canadian authorities are also investigating other cattle that are at “equivalent risk.” Those animals will be killed and then tested for BSE. Currently, there is no reliable way to test a live cow for BSE. Instead, scientists look at the animal’s brain tissue under a microscope to see if it has a spongy appearance. If so, the cow is confirmed with the disease.

Symptoms of BSE include lack of coordination, abnormal posture, or nervous or violent behavior, therefore the nickname “mad cow.” One of the key issues with diagnosing the disease early is that the symptoms can lay dormant for four to six years after the animal contracts BSE.

The World Health Organization for Animal Health rates Canada as a “controlled BSE risk” country, and industry sources said the current case is unlikely to change that.

© Food Safety News

    Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of
    Animals and Animal Products Singeltary Submission ;

    I believe that there is more risk to the world from Transmissible
    Spongiform Encephalopathy TSE prion aka mad cow type disease now, coming from
    the United States and all of North America, than there is risk coming to the USA
    and North America, from other Countries. I am NOT saying I dont think there is
    any risk for the BSE type TSE prion coming from other Countries, I am just
    saying that in 2015, why is the APHIS/USDA/FSIS/FDA still ignoring these present
    mad cow risk factors in North America like they are not here?

    North America has more strains of TSE prion disease, in more species
    (excluding zoo animals in the early BSE days, and excluding the Feline TSE and
    or Canine TSE, because they dont look, and yes, there has been documented
    evidence and scientific studies, and DEFRA Hound study, that shows the canine
    spongiform encephalopathy is very possible, if it has not already happened, just
    not documented), then any other Country in the world. Mink TME, Deer Elk cervid
    CWD (multiple strains), cBSE cattle, atypical L-type BSE cattle, atypical H-type
    BSE cattle, atyical HG type BSE cow (the only cow documented in the world to
    date with this strain), typical sheep goat Scrapie (multiple strains), and the
    atypical Nor-98 Scrapie, which has been linked to sporadic CJD, Nor-98 atypical
    Scrapie has spread from coast to coast. sporadic CJD on the rise, with different
    strains mounting, victims becoming younger, with the latest nvCJD human mad cow
    case being documented in Texas again, this case, NOT LINKED TO EUROPEAN TRAVEL

    typical BSE can propagate as nvCJD and or sporadic CJD (Collinge et al),
    and sporadic CJD has now been linked to atypical BSE, Scrapie and atypical
    Scrapie, and scientist are very concerned with CWD TSE prion in the Cervid
    populations. in my opinion, the BSE MRR policy, which overtook the BSE GBR risk
    assessments for each country, and then made BSE confirmed countries legal to
    trade mad cow disease, which was all brought forth AFTER that fateful day
    December 23, 2003, when the USA lost its gold card i.e. BSE FREE status, thats
    the day it all started. once the BSE MRR policy was shoved down every countries
    throat by USDA inc and the OIE, then the legal trading of Scrapie was validated
    to be a legal trading commodity, also shoved through by the USDA inc and the
    OIE, the world then lost 30 years of attempted eradication of the BSE TSE prion
    disease typical and atypical strains, and the BSE TSE Prion aka mad cow type
    disease was thus made a legal trading commodity, like it or not. its all about
    money now folks, trade, to hell with human health with a slow incubating
    disease, that is 100% fatal once clinical, and forget the fact of exposure,
    sub-clinical infection, and friendly fire there from i.e. iatrogenic TSE prion
    disease, the pass it forward mode of the TSE PRION aka mad cow type disease. its
    all going to be sporadic CJD or sporadic ffi, or sporadic gss, or now the
    infamous VPSPr. …problem solved $$$

    the USDA/APHIS/FSIS/FDA triple mad cow BSE firewall, well, that was nothing
    but ink on paper.

    for this very reason I believe the BSE MRR policy is a total failure, and
    that this policy should be immediately withdrawn, and set back in place the BSE
    GBR Risk Assessments, with the BSE GBR risk assessments set up to monitor all
    TSE PRION disease in all species of animals, and that the BSE GBR risk
    assessments be made stronger than before.

    lets start with the recent notice that beef from Ireland will be coming to

    Ireland confirmed around 1655 cases of mad cow disease. with the highest
    year confirming about 333 cases in 2002, with numbers of BSE confirmed cases
    dropping from that point on, to a documentation of 1 confirmed case in 2013, to
    date. a drastic decrease in the feeding of cows to cows i.e. the ruminant mad
    cow feed ban, and the enforcement of that ban, has drastically reduced the
    number of BSE cases in Europe, minus a few BABs or BARBs. a far cry from the
    USDA FDA triple BSE firewall, which was nothing more than ink on paper, where in
    2007, in one week recall alone, some 10 MILLION POUNDS OF BANNED POTENTIAL MAD
    COW FEED WENT OUT INTO COMMERCE IN THE USA. this is 10 years post feed ban. in
    my honest opinion, due to the blatant cover up of BSE TSE prion aka mad cow
    disease in the USA, we still have no clue as to the true number of cases of BSE
    mad cow disease in the USA or North America as a whole. …just saying.

    Number of reported cases of bovine spongiform encephalopathy (BSE) in
    farmed cattle worldwide* (excluding the United Kingdom)


    snip…please see attached pdf file, with references of breaches in the USA
    triple BSE mad cow firewalls, and recent science on the TSE prion disease.

    No documents available.

    AttachmentsView All (1)


    Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of
    Animals and Animal Products Singeltary Submission

    View Attachment:

    Singeltary Submission to USDA 2014 BSE CJD TSE PRION


    Sunday, January 11, 2015

    Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of
    Animals and Animal Products Singeltary Submission


    Discussion: The C, L and H type BSE cases in Canada exhibit molecular
    characteristics similar to those described for classical and atypical BSE cases
    from Europe and Japan. *** This supports the theory that the importation of BSE
    contaminated feedstuff is the source of C-type BSE in Canada. *** It also
    suggests a similar cause or source for atypical BSE in these countries. ***

    see page 176 of 201 pages…tss


    *** PLOS Singeltary reply ; Molecular, Biochemical and Genetic
    Characteristics of BSE in Canada Singeltary reply ;


    PLOS Singeltary Comment ;

    ruminant feed ban for cervids in the United States ?

    31 Jan 2015 at 20:14 GMT


    Saturday, January 24, 2015

    Bovine Spongiform Encephalopathy: Atypical Pros and Cons


    Saturday, January 31, 2015

    RAPID ADVICE 17-2014 : Evaluation of the risk for public health of casings
    in countries with a “negligible risk status for BSE” and on the risk of
    modification of the list of specified risk materials (SRM) with regard to BSE


    Friday, January 30, 2015

    Scrapie: a particularly persistent pathogen


    Tuesday, December 16, 2014

    Evidence for zoonotic potential of ovine scrapie prions

    Hervé Cassard,1, n1 Juan-Maria Torres,2, n1 Caroline Lacroux,1, Jean-Yves
    Douet,1, Sylvie L. Benestad,3, Frédéric Lantier,4, Séverine Lugan,1, Isabelle
    Lantier,4, Pierrette Costes,1, Naima Aron,1, Fabienne Reine,5, Laetitia
    Herzog,5, Juan-Carlos Espinosa,2, Vincent Beringue5, & Olivier Andréoletti1,
    Affiliations Contributions Corresponding author Journal name: Nature
    Communications Volume: 5, Article number: 5821 DOI: doi:10.1038/ncomms6821
    Received 07 August 2014 Accepted 10 November 2014 Published 16 December 2014
    Article tools Citation Reprints Rights & permissions Article metrics


    Although Bovine Spongiform Encephalopathy (BSE) is the cause of variant
    Creutzfeldt Jakob disease (vCJD) in humans, the zoonotic potential of scrapie
    prions remains unknown. Mice genetically engineered to overexpress the human
    ​prion protein (tgHu) have emerged as highly relevant models for gauging the
    capacity of prions to transmit to humans. These models can propagate human
    prions without any apparent transmission barrier and have been used used to
    confirm the zoonotic ability of BSE. Here we show that a panel of sheep scrapie
    prions transmit to several tgHu mice models with an efficiency comparable to
    that of cattle BSE. The serial transmission of different scrapie isolates in
    these mice led to the propagation of prions that are phenotypically identical to
    those causing sporadic CJD (sCJD) in humans. These results demonstrate that
    scrapie prions have a zoonotic potential and raise new questions about the
    possible link between animal and human prions.

    Subject terms: Biological sciences• Medical research At a glance


    why do we not want to do TSE transmission studies on chimpanzees $

    5. A positive result from a chimpanzee challenged severly would likely
    create alarm in some circles even if the result could not be interpreted for
    man. I have a view that all these agents could be transmitted provided a large
    enough dose by appropriate routes was given and the animals kept long enough.
    Until the mechanisms of the species barrier are more clearly understood it might
    be best to retain that hypothesis.




    CDC ***

    Sunday, November 23, 2014

    *** Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in
    Texas in June 2014 confirmed as USA case NOT European ***

    the patient had resided in Kuwait, Russia and Lebanon. The completed
    investigation did not support the patient’s having had extended travel to
    European countries, including the United Kingdom, or travel to Saudi Arabia. The
    specific overseas country where this patient’s infection occurred is less clear
    largely because the investigation did not definitely link him to a country where
    other known vCJD cases likely had been infected.


    Sunday, December 14, 2014

    *** ALERT new variant Creutzfeldt Jakob Disease nvCJD or vCJD, sporadic
    CJD strains, TSE prion aka Mad Cow Disease United States of America Update
    December 14, 2014 Report ***


    UPDATE* NOVEMBER 16, 2014

    vpspr, sgss, sffi, TSE, an iatrogenic by-product of gss, ffi, familial
    type prion disease, what it ???

    Friday, January 10, 2014

    Greetings again Friends, Neighbors, and Colleagues,

    I would kindly like to follow up on ‘vpspr, sgss, sffi, TSE, an iatrogenic
    by-product of gss, ffi, familial type prion disease, what it ???’ ran across an
    old paper from 1984, that some might find interest in, and I will update the
    link with this old science paper from 1984, a 2010 paper from Japan, and some
    information on scrapie transmission. The paper from Japan first, then the 1984
    paper, and then the scrapie transmission studies.

    ***The occurrence of contact cases raises the possibility that
    transmission in families may be effected by an unusually virulent strain of the


    Subject: *** Becky Lockhart 46, Utah’s first female House speaker, dies
    diagnosed with the extremely rare Creutzfeldt-Jakob disease aka mad cow type

    what is CJD ? just ask USDA inc., and the OIE, they are still feeding the
    public and the media industry fed junk science that is 30 years old.

    why doesn’t some of you try reading the facts, instead of rubber stamping
    everything the USDA inc says.

    sporadic CJD has now been linked to BSE aka mad cow disease, Scrapie, and
    there is much concern now for CWD and risk factor for humans.

    My sincere condolences to the family and friends of the House Speaker
    Becky Lockhart. I am deeply saddened hear this.

    with that said, with great respect, I must ask each and every one of you
    Politicians that are so deeply saddened to hear of this needless death of the
    Honorable House Speaker Becky Lockhart, really, cry me a friggen river. I am
    seriously going to ask you all this…I have been diplomatic for about 17 years
    and it has got no where. people are still dying. so, are you all stupid or
    what??? how many more need to die ??? how much is global trade of beef and other
    meat products that are not tested for the TSE prion disease, how much and how
    many bodies is this market worth?

    Saturday, January 17, 2015

    *** Becky Lockhart 46, Utah’s first female House speaker, dies diagnosed
    with the extremely rare Creutzfeldt-Jakob disease


    Saturday, February 14, 2015

    *** Canadian Food Inspection Agency Confirms Bovine Spongiform
    Encephalopathy (BSE) in Alberta


    Tuesday, February 10, 2015

    *** Alberta Canada First case of chronic wasting disease found in farm elk
    since 2002


    Terry S. Singeltary Sr.

    • Veterinary Record 2015;176:159-160 doi:10.1136/vr.h784

      Could we spot the next BSE?, asks BVA President

      CONCERN about the robustness of the surveillance network in England and
      Wales was expressed by the BVA President, John Blackwell, in his speech to the
      Association’s annual London dinner last week.

      Mr Blackwell said that, while the BVA understood the need for
      rationalisation and efficiency, it was concerned that the surveillance system
      that had been relied on in recent years was being dismantled without the
      replacement being properly tested. If information coming from postmortem
      examinations was not systematically and consistently fed into a central data
      collection point, it would be ‘a lot harder to join the dots’ and to spot a
      problem, something that was the ‘very foundation of a robust surveillance

      ‘If there is now a risk that we have a less responsive and accurate
      diagnosis system, a system that is as yet not joined up and integrated, we leave
      ourselves vulnerable, less able to spot new and emerging diseases and act
      quickly to contain them’

      As well as identifying known threats, a robust surveillance mechanism
      needed to identify the unknowns: ‘If there is now a risk that we have a less
      responsive and accurate diagnosis system, a system that is as yet not joined up
      and integrated, we leave ourselves vulnerable, less able to spot new and
      emerging diseases and act quickly to contain them,’ said Mr Blackwell. ‘This
      risk is multiplied if the network of surveillance – that strategic ability to
      horizon scan – is patchy. We fear this may now be the case. Soon after I
      qualified back in 1985, BSE was effectively diagnosed because of our network of
      surveillance laboratories. A network that allowed us to grasp and understand the
      emerging threat and identify the unknown risk. Are we confident we have the
      systems in place to spot the next emergent threat, the next …


      > Could we spot the next BSE?

      we have not spotted all the cases the first time around. with Nations like
      the United States and Canada, organizations like the USDA, OIE, and WTO et al,
      it was never about ‘spotting’ all the BSE TSE prion cases, it was more about how
      not to find them. the triple BSE mad cow firewall, was and still is, nothing but
      ink on paper. …please see facts ;

      Tuesday, February 17, 2015

      Could we spot the next BSE?, asks BVA President