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Colbert Grills AMI on Meat Safety (Video)

American Meat Institute President J. Patrick Boyle brought meat regulatory issues into the spotlight on Comedy Central’s Emmy award-winning Colbert Report Tuesday night. Boyle appeared on the show for part 12 of Stephen Colbert’s “35,000-part series,” dubbed “Know Your Lobby,” which grills Washington lobbyists.

The hilarious four-minute segment–which discusses food safety, meat consumption, and cannibalism–was based off a grueling two-hour interview with Boyle, the lead spokesman for 95 percent of the American meat industry.

“This country was built on meat,” proclaims master satirist Colbert at the beginning of the segment. “Its been a part of our history ever since the declaration of our independence was branded on a flank steak. Check the signatures closely, you’ll see one of them is Jimmy Dean.”

Colbert hits AMI, the “fightin’ meat men,” as he calls them, over the use ammonia for food safety and for opposing the regulation on six non-O157 strains of E. coli.

“One way the AMI is looking out for you is by defending sterilizing beef with ammonia,” says Colbert, hinting at the recent controversy over a New York Times expose on the practice. “So not only will your beef be disease-free, once it passes through you it will leave your toilet with a mirror shine.”

“The AMI recently opposed a bill introduced by Senator Kirsten Gillibrand [D-NY] that would require the USDA to regulate six additional strains of disease-causing E. coli. Bravo,” says faux-conservative Colbert. “I say we let the market regulate E. coli, because if you get a burger with E. coli, you’ll never buy that hamburger again.”

“I would not encourage you to eat any strain of E. coli,” says Boyle, who begins to explain why AMI opposes Gillibrand’s bill but is abruptly cut off by Colbert.

The food safety exchange continues and Colbert asks, “Should I be able to get my burger rare?”

“I would not order my burger rare,” says Boyle.

“I don’t want you to take this as an insult, because it’s not meant that way, it’s just an observation, but you sound like a coward,” Colbert retorts.

“Although I’ve had a lot of tough interviews in my day, that was definitely the toughest and most unique interview experience of my career,” Boyle said in an AMI statement yesterday. “I was honored to play straight man to the hilarious Stephen Colbert and to demonstrate that while meat processing is serious business, we can be good sports.”

You can watch the full segment here:

© Food Safety News
  • Paul Nunes

    Brilliant!

  • http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html Terry S. Singeltary Sr.

    let’s take a closer look at this new prionpathy or prionopathy, and then let’s look at the g-h-BSEalabama mad cow.
    This new prionopathy in humans? the genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_ mad cow in the world to date like this, ……wait, it get’s better. this new prionpathy is killing young and old humans, with LONG DURATION from onset of symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and the plaques are very similar in some cases too, bbbut, it’s not related to the g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that they claim is a genetic TSE, has no relation to any gene mutation in that family. daaa, ya think it could be related to that mad cow with the same genetic make-up ??? there were literally tons and tons of banned mad cow protein in Alabama in commerce, and none of it transmitted to cows, and the cows to humans there from ??? r i g h t $$$
    Rural and Regional Affairs and Transport References Committee The possible impacts and consequences for public health, trade and agriculture of the Government’s decision to relax import restrictions on beef Final report June 2010
    2.65 At its hearing on 14 May 2010, the committee heard evidence from Dr Alan Fahey who has recently submitted a thesis on the clinical neuropsychiatric, epidemiological and diagnostic features of Creutzfeldt-Jakob disease.48 Dr Fahey told the committee of his concerns regarding the lengthy incubation period for transmissible spongiform encephalopathies, the inadequacy of current tests and the limited nature of our current understanding of this group of diseases.49
    2.66 Dr Fahey also told the committee that in the last two years a link has been established between forms of atypical CJD and atypical BSE. Dr Fahey said that: They now believe that those atypical BSEs overseas are in fact causing sporadic Creutzfeldt-Jakob disease. They were not sure if it was due to mad sheep disease or a different form. If you look in the textbooks it looks like this is just arising by itself. But in my research I have a summary of a document which states that there has never been any proof that sporadic Creutzfeldt-Jakob disease has arisen de novo—has arisen of itself. There is no proof of that. The recent research is that in fact it is due to atypical forms of mad cow disease which have been found across Europe, have been found in America and have been found in Asia. These atypical forms of mad cow disease typically have even longer incubation periods than the classical mad cow disease.50
    http://www.aph.gov.au/senate/committee/rrat_ctte/mad_cows/report/report.pdf
    ALABAMA MAD COW g-h-BSEalabama
    In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in “the approximately 10-year-old cow” carrying the E221K mutation.
    http://www.plospathogens.org/article/info%3Adoi%2F10.1371%2Fjournal.ppat.1000156
    http://www.plospathogens.org/article/fetchObjectAttachment.action?uri=info%3Adoi%2F10.1371%2Fjournal.ppat.1000156&representation=PDF
    Saturday, August 14, 2010
    BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY
    (see mad cow feed in COMMERCE IN ALABAMA…TSS)
    http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html
    g-h-BSE-alabama E211K mad cows USA how many would that be annually ???
    if our ciphering is correct (?), that would be about 35 g-h-BSE-alabama E211K mad cows going into the food chain a year.
    an incidence of less than 1 in 2000.
    let’s see, that’s 500 such per million.
    or 50,000 cows per 100 million (US herd size).
    even at less than 1 in a million, with 35 million slaughtered, that’s 35 infected cows going into the food chain each year.
    hmmm, friendly fire there from ???
    Wednesday, July 28, 2010
    re-Freedom of Information Act Project Number 3625-32000-086-05, Study of Atypical BSE UPDATE July 28, 2010
    http://bse-atypical.blogspot.com/2010/07/re-freedom-of-information-act-project.html
    Tuesday, August 03, 2010
    Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein
    http://creutzfeldt-jakob-disease.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html
    Monday, August 9, 2010
    Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein or just more PRIONBALONEY ?
    http://prionunitusaupdate2008.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html
    Thursday, August 12, 2010
    Seven main threats for the future linked to prions
    http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html
    http://prionpathy.blogspot.com/
    Tuesday, March 16, 2010
    COMMONWEALTH OF AUSTRALIA Hansard Import restrictions on beef FRIDAY, 5 FEBRUARY 2010 AUSTRALIA
    COMMONWEALTH OF AUSTRALIA
    Proof Committee Hansard
    RRA&T 2 Senate Friday, 5 February 2010
    RURAL AND REGIONAL AFFAIRS AND TRANSPORT
    [9.03 am]
    BELLINGER, Mr Brad, Chairman, Australian Beef Association
    CARTER, Mr John Edward, Director, Australian Beef Association
    CHAIR—Welcome. Would you like to make an opening statement?
    Mr Bellinger—Thank you. The ABA stands by its submission, which we made on 14 December last year, that the decision made by the government to allow the importation of beef from BSE affected countries is politically based, not science based. During this hearing we will bring forward compelling new evidence to back up this statement. When I returned to my property after the December hearing I received a note from an American citizen. I will read a small excerpt from the mail he sent me in order to reinforce the dangers of allowing the importation of beef from BSE affected countries. I have done a number of press releases on this topic, and this fellow has obviously picked my details up from the internet. His name is Terry Singeltary and he is from Bacliff, Texas. He states, and rightfully so:
    You should be worried. Please let me explain. I’ve kept up with the mad cow saga for 12 years today, on December 14th 1997, some four months post voluntary and partial mad cow feed ban in the USA, I lost my mother to the Heinemann variant Creutzfeldt-Jakob disease (CJD). I know this is just another phenotype of the infamous sporadic CJDs. Here in the USA, when USA sheep scrapie was transmitted to USA bovine, the agent was not UK BSE—it was a different strain. So why then would human TSE from USA cattle look like UK CJD from UK BSE? It would not. So this accentuates that the science is inconclusive still on this devastating disease. He goes on to state:
    The OIE— the International Organisation of Epizootics, the arm of the WTO— is a failed global agent that in my opinion is bought off via bogus regulations for global trade and industry reps. I have done this all these years for nothing but the truth. I am a consumer, I eat meat, but I do not have to sit idly by and see the ignorance and greed of it all while countless numbers of humans and animals are being exposed to the TSE agents. All the USA is interested in is trade, nothing else matters.
    Even Dr Stanley Prusiner, who incidentally won the Nobel Health Prize in 1997 for his work on the prion—he invented the word ‘prion’, or it came from him—states:
    The BSC policy was set up for one purpose only, trade—the illegal trading of all strains of TSE globally throughout North America, which is home to CBSC, IBSC and HBSC, many scrapie strains and two strains of CJD to date. (please note typo error, those should have read cBSE, lBSE, and hBSE…tss)
    I would also like, while I have the opportunity, to explain the beef-off-the-shelves myth. At the first Senate hearing on 14 December, it was explained that the reason why they allowed BSC beef into Australia was the beef-off-the-shelves policy, whereby if we found a case of BSC in Australia they would have to recall all—
    Friday, 5 February 2010 Senate RRA&T 3
    RURAL AND REGIONAL AFFAIRS AND TRANSPORT
    Senator HEFFERNAN—Which of course is total BS.
    Mr Bellinger—Correct. This is written in the FSANZ document—Food Standards Australia New Zealand. Why isn’t this same policy in New Zealand? It is not—it is only in Australia. We are the only country in the world to have this idiotic policy. So we again call for the tabling of the WTO obligations paperwork. We do not believe that exists.
    snip…see full text 110 pages ;
    http://www.aph.gov.au/hansard/senate/commttee/S12742.pdf
    for those interested, please see much more here ;
    http://docket-aphis-2006-0041.blogspot.com/2010/03/commonwealth-of-australia-hansard.html
    http://transmissiblespongiformencephalopathy.blogspot.com/2010/02/transmissible-spongiform-encephalopathy.html
    Tuesday, July 13, 2010
    (SEE BEEF PRODUCTS EXPORTED TO AUSTRALIA FROM USA…TSS)
    AUSTRALIAN QUESTIONNAIRE TO ASSESS BSE RISK (OIE) Terrestrial Animal Health Code, 2009 and USA export risk factor for BSE to Australia
    http://usdameatexport.blogspot.com/2010/07/australian-questionnaire-to-assess-bse.html
    Saturday, August 14, 2010
    USA NON-SPECIES CODING SYSTEM (BEEF IMPORT EXPORT BSE RISK THERE FROM)
    US denies it’s illegally sending beef to Australia ?
    Friday, 13/08/2010
    http://usdameatexport.blogspot.com/2010/08/usa-non-species-coding-system-beef.html
    Saturday, June 19, 2010
    U.S. DENIED UPGRADED BSE STATUS FROM OIE
    http://usdameatexport.blogspot.com/2010/06/us-denied-upgraded-bse-status-from-oie.html
    Sunday, August 15, 2010
    ATYPICAL BSE NOW LINKED TO CAUSING SPORADIC CJD OVERSEAS Commonwealth of Australia
    http://bse-atypical.blogspot.com/2010/08/atypical-bse-now-linked-to-causing.html
    Tuesday, July 27, 2010
    Spontaneous generation of mammalian prions
    http://madcowspontaneousnot.blogspot.com/2010/07/spontaneous-generation-of-mammalian.html
    P.9.21
    Molecular characterization of BSE in Canada
    Jianmin Yang1, Sandor Dudas2, Catherine Graham2, Markus Czub3, Tim McAllister1, Stefanie Czub1 1Agriculture and Agri-Food Canada Research Centre, Canada; 2National and OIE BSE Reference Laboratory, Canada; 3University of Calgary, Canada
    Background: Three BSE types (classical and two atypical) have been identified on the basis of molecular characteristics of the misfolded protein associated with the disease. To date, each of these three types have been detected in Canadian cattle.
    Objectives: This study was conducted to further characterize the 16 Canadian BSE cases based on the biochemical properties of there associated PrPres. Methods: Immuno-reactivity, molecular weight, glycoform profiles and relative proteinase K sensitivity of the PrPres from each of the 16 confirmed Canadian BSE cases was determined using modified Western blot analysis.
    Results: Fourteen of the 16 Canadian BSE cases were C type, 1 was H type and 1 was L type. The Canadian H and L-type BSE cases exhibited size shifts and changes in glycosylation similar to other atypical BSE cases. PK digestion under mild and stringent conditions revealed a reduced protease resistance of the atypical cases compared to the C-type cases. N terminal- specific antibodies bound to PrPres from H type but not from C or L type. The C-terminal-specific antibodies resulted in a shift in the glycoform profile and detected a fourth band in the Canadian H-type BSE.
    Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. It also suggests a similar cause or source for atypical BSE in these countries.
    http://www.prion2009.com/sites/default/files/Prion2009_Book_of_Abstracts.pdf
    Wednesday, August 11, 2010
    REPORT ON THE INVESTIGATION OF THE SIXTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA
    http://bse-atypical.blogspot.com/2010/08/report-on-investigation-of-sixteenth.html
    Thursday, August 19, 2010
    REPORT ON THE INVESTIGATION OF THE SEVENTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA
    http://bseusa.blogspot.com/2010/08/report-on-investigation-of-seventeenth.html
    Thursday, August 19, 2010
    SCRAPIE CANADA UPDATE Current as of 2010-07-31 The following table lists sheep flocks and/or goat herds confirmed to be infected with scrapie in Canada in 2010.
    Current as of: 2010-07-31
    http://nor-98.blogspot.com/2010/08/scrapie-canada-update-current-as-of.html
    14th ICID International Scientific Exchange Brochure -
    Final Abstract Number: ISE.114
    Session: International Scientific Exchange
    Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America
    update October 2009
    T. Singeltary
    Bacliff, TX, USA
    Background:
    An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie’s, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.
    Methods:
    12 years independent research of available data
    Results:
    I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.
    Conclusion:
    I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena’s. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.
    http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf
    Wednesday, March 31, 2010
    Atypical BSE in Cattle
    http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html
    FRIENDLY FIRE FROM ALL OF THE ABOVE ;
    Wednesday, August 18, 2010
    Incidence of CJD Deaths Reported by CJD-SS in Canada as of July 31, 2010
    http://creutzfeldt-jakob-disease.blogspot.com/2010/08/incidence-of-cjd-deaths-reported-by-cjd.html
    Monday, August 9, 2010
    National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)
    (please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)
    http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html
    Thursday, August 12, 2010
    USA Blood products, collected from a donor who was at risk for vCJD, were distributed July-August 2010
    http://creutzfeldt-jakob-disease.blogspot.com/2010/08/usa-blood-products-collected-from-donor.html
    Sunday, August 8, 2010
    The Transcellular Spread of Cytosolic Amyloids, Prions, and Prionoids
    http://betaamyloidcjd.blogspot.com/2010/08/transcellular-spread-of-cytosolic.html
    Sunday, July 18, 2010
    Alzheimer’s Assocition International Conference on Alzheimer’s Disease (updated diagnostic criteria) 2010 July 10 – 15 Honolulu, Hawaii
    http://betaamyloidcjd.blogspot.com/2010/07/alzheimers-assocition-international.html
    http://betaamyloidcjd.blogspot.com/
    Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518 flounder9@verizon.net