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Is American Meat Unsafe or is this a Trade War?

Is “Made in the U.S.A.” safe?  Or, is this a trade war?

When I was a kid in the early 1960s, I recall that “Made in Japan” meant cheap, but also sub-par in quality.  Interesting how that has changed (compare Japanese made cars to those made in the U.S. for quality and sales).

More recently, in my world we continue to hear people voice a fear of food made “overseas”–in China or Mexico–that is somehow supposed to be unsafe, or less safe than food produced in the U.S.  Yes, there have been instances of foreign food products sickening Americans (melamine in dog and cat food from China, Salmonella in cantaloupes from Honduras, hepatitis A in green onions from Mexico), but in 17 years of being involved in litigation resulting from nearly every foodborne illness outbreak in the U.S., in my experience most of the food products that sicken us are home-grown and mass-produced.  As I have said more that a few times, “U.S. corporations do a marvelous job of poisoning us.”

It seems that other countries are now paying attention to what we sell (or try to sell) them.  Perhaps, like me in the 1960s, they think “Made in the USA” means something far different that what the producers and manufacturers would wish.  

Look at the recent dust-up over chicken in Russia.  Russia may stop importing poultry by 2015, Prime Minister Vladimir Putin said recently, backing a ban imposed on U.S. chicken imports at the beginning of the year.   “We haven’t seen any readiness to meet Russian standards on the part of some of our partners, mainly the companies from the United States,” he said.

“If our foreign suppliers are unable or reluctant to meet our security requirements, we will use other sources.”

Perhaps Putin is playing to the Russian poultry industry (goodness, we never see U.S. politicians doing the same here), or, perhaps he read the study released by Consumer Reports that found two-thirds of fresh broiler chicken purchased harbored Salmonella and/or Campylobacter.  The study also found that most of the bacteria sampled from the raw chicken was resistant to at least one antibiotic.

And, what about Asia?  In the latest controversy over “Made in the U.S.A.,” in late December, Taiwan’s ruling and opposition parties reached an agreement to amend the island nation’s Food Sanitation Act to bar the import of bone-in and certain other beef products from the U.S. for fear of Mad Cow disease.

True or not, can you blame them for being a bit worried over U.S. beef after reading about the use of ammonia-treated meat, a substance compared to “pink slime”, in hamburgers served at fast food restaurants and through the United States Department of Agriculture’s (USDA’s) National School Lunch Program?

Carl Custer, a former USDA microbiologist, told the New York Times that he and other scientists were concerned about the potential safety risks associated with the consumption of ammonia-treated beef without independent validation of the meat’s safety.  “I do not consider the stuff to be ground beef, and I consider allowing it in ground beef to be a form of fraudulent labeling,” he told the Times.

Trade war? Perhaps.  Or not. However, you must admit that the meat industry and our own government are handing the stick to allow foreign politicians to beat the meat industry in the head.  

Seriously, we have Salmonella and Campylobacter found in a large percentage of chicken in our stores, and a “pink slime” being served to our school children in ground beef–“ground beef” that a former USDA employee did not even consider to be ground beef–“ground beef” that contains “pink slime” but is not labeled as containing ammonia or ammonia-treated beef as an ingredient, which caused the same former government employee to consider omission of ammonia on food labels as “fraudulent.”

“Made in USA” used to mean something different.

© Food Safety News
  • You wrote; Taiwan’s ruling and opposition parties reached an agreement to amend the island nation’s Food Sanitation Act to bar the import of bone-in and certain other beef products from the U.S. for fear of Mad Cow disease (BSE)….
    However, BSE can be a naturally occurring disease, so not an infectious disease. WHY? Because, about the BSE disease; this was never justified scientifically! It was pure, math-model-driven science fiction. But it was pushed very vigorously by the British science establishment, which has never confessed to its errors, and is therefore likely to make the same ones again.
    See recently; the swine flu outbreak was a ‘false pandemic’; said Wolfgang Wodarg, head of health at the Council of Europe (January 8, 2010). He has branded the H1N1 outbreak as ‘one of the greatest medical scandals of the century’!
    However, the pandemic has started on May 1, 2009 when Roy Anderson was interviewed on the BBC´s Today programme about the 2009 swine flu outbreak. He was a member of a group set up to give scientific advice to the British government over health issues relating to swine flu. However, swine flu is not the first time we have suffered this nonsense. See predictions about BSE/ vCJD by Roy M. Anderson. He has mathematically modelled the spread of new variant Creutzfeld- Jakob disease (v CJD), published in Nature (406, 583-584; 10 August 2000).
    There his team showed that the current mortality data are consistent with between 63 and 136,000 cases among the population known to have a susceptible genotype (about 40% of the total population), with on average less than two cases of vCJD arising from the consumption of one infected bovine. However, far fewer people are carrying the human form of mad cow disease than previously feared. There have been (to date) 168 definite or probable cases of vCJD since 1995, suggesting that the risk had been „overestimated“.
    This led to an obscene £5 billion campaign of British cattle destruction and compensation. And about the USA ? The International Trade Commission released a report estimating that trade restrictions resulting from BSE cost the cattle industry $11 billion from 2004 to 2007
    (www.beefusa.org/NEWSTradeRestrictionsCostCattlemen11BillioninLostSales36991.aspx). This could be “more greatest medical scandal”!
    See other relationships, according to my recent presentation at 29th World Veterinary Congress in Vancouver; Neurodegenerative Diseases and Schizophrenia as a Hyper or Hypofunction of the NMDA Receptors (www.bse-expert.cz/pdf/Veter_kongres.pdf).
    Sincerely
    Josef Hlasny, DVM, PhD., veterinary surgeon in Bludov, Czech Republic

  • Continued…In Britain, much of the alarmism about Mad Cow disease was never justified scientifically. It was pure, math-model-driven science fiction… There are some; „eight BSE/ vCJD mathematical examples“, mostly headed by R.G.WILL (National Creutzfeldt-Jakob Disease Surveillance Unit in Edinburgh):
    1. Dev Biol Stand. 1998;93:79-84.
    New variant Creutzfeldt-Jakob disease.
    Will RG.
    National Creutzfeldt-Jakob Disease Surveillance Unit, Western General Hospital, Edinburgh, UK.
    New variant Creutzfeldt-Jakob disease is a novel human spongiform encephalopathy with a consistent clinico-pathological phenotype. Epidemiological evidence indicates that this disease is occurring almost exclusively in the UK, where there has been an epidemic of spongiform encephalopathy in the cattle population. Current evidence strongly supports the hypothesis that there is a causal link between bovine spongiform encephalopathy and new variant Creutzfeldt-Jakob disease.
    2. Nature 406, 583-584 (10 August 2000) | doi:10.1038/35020688
    Predicted vCJD mortality in Great Britain
    Azra C. Ghani1, Neil M. Ferguson1, Christl A. Donnelly1 & Roy M. Anderson1
    Top of page
    Abstract
    Modelling the latest data puts a ceiling on the likely number of vCJD cases.
    Top of page
    Abstract
    There is continued speculation about the likely number of cases of variant Creutzfeldt–Jakob disease (vCJD) that will occur in Great Britain in the wake of the BSE epidemic in cattle and in light of a recent cluster of vCJD cases in Leicestershire, England. We show here that the current mortality data are consistent with between 63 and 136,000 cases among the population known to have a susceptible genotype (about 40% of the total population), with on average less than two cases of vCJD arising from the consumption of one infected bovine.
    3. Lancet. 2000 Aug 5;356(9228):481-2.
    Incidence of variant Creutzfeldt-Jakob disease in the UK.
    Andrews NJ, Farrington CP, Cousens SN, Smith PG, Ward H, Knight RS, Ironside JW, Will RG.
    The number of deaths from variant CJD (vCJD) in the UK increased in the last quarter of 1998, although numbers were lower in subsequent quarters. We analysed the numbers of definite and probable (living and dead) vCJD cases since 1994 to assess trends in incidence. We estimated that the number of onsets increased by 23% per year for 1994-2000 (p=0.004), and that deaths increased by 33% for 1995-2000 (p=0.005). The absolute number of cases in the UK is still low, but such an increase should be a matter of concern.
    4. Science. 2001 Nov 23;294(5547):1729-31. Epub 2001 Oct 25.
    Predictability of the UK variant Creutzfeldt-Jakob disease epidemic.
    d’Aignaux JN, Cousens SN, Smith RG.
    London School of Hygiene and Tropical Medicine, Infectious Disease Epidemiology Unit, Keppel Street, London WC1E 7HT, UK. jerome.huillard@lshtm.ac.uk
    Comment in: Science. 2001 Nov 23;294(5547):1663-4.
    Back-calculation analysis of the variant Creutzfeldt-Jakob disease epidemic in the United Kingdom is used to estimate the number of infected individuals and future disease incidence. The model assumes a hazard of infection proportional to the incidence of bovine spongiform encephalopathy in the United Kingdom and accounts for precautionary control measures and very wide ranges of incubation periods. The model indicates that current case data are compatible with numbers of infections ranging from a few hundred to several millions. In the latter case, the model suggests that the mean incubation period must be well beyond the human life-span, resulting in
    5. Lancet. 2003 Mar 1;361(9359):751-2.
    Deaths from variant Creutzfeldt-Jakob disease in the UK.
    Andrews NJ, Farrington CP, Ward HJ, Cousens SN, Smith PG, Molesworth AM, Knight RS, Ironside JW, Will RG.
    Public Health Laboratory Service, Communicable Disease Surveillance Centre, London, UK.
    In 2002, 17 people died from variant CJD (vCJD) in the UK, compared with 20 in 2001 and 28 in 2000. We analysed data for deaths from vCJD since 1995 and estimated the underlying trend in mortality. The trend had a quadratic component (p=0.005), suggesting that the increase was not exponential, and that the previously increasing trend is slowing down. The death rate peaked in 2000. These findings are encouraging, but mortality might increase again in the future.
    6. Stat Methods Med Res. 2003 Jun;12(3):203-20.
    The predictability of the epidemic of variant Creutzfeldt-Jakob disease by back-calculation methods.
    Huillard d’Aignaux JN, Cousens SN, Smith R,G.
    Department of Infectious and Tropical Diseases, London School of Hygiene and Tropical Medicine, London, UK.
    We present a back-calculation analysis of the variant Creutzfeldt-Jakob (vCJD) epidemic in the UK to estimate the number of infected individuals and to explore the likely future incidence of the disease. The main features of the model are that the hazard of infection was assumed proportional to the incidence of BSE in the UK with allowance for precautionary control measures taken in 1988 and in 1996, and that the incubation period distribution of vCJD follows an offset generalized F distribution. Our results indicate that current the numbers of cases with onset up to 31 December 2000 data are broadly compatible with numbers of primary infections ranging from a few hundred to several million. However, if a very large number of persons were infected, the model suggests that the mean incubation period is likely to be well beyond the human lifespan, resulting in a disease epidemic of much smaller size (maximum several thousand). A sensitivity analysis indicates that our results are sensitive to the underreporting of vCJD cases before 1996. Finally, we show that, in the absence of a reliable test for asymptomatic infection, uncertainty in estimates of the total number of infections is likely to remain for at least several years, even if the number of clinical cases remains low.
    7. Curr Top Microbiol Immunol.
    2004;284:161-91.
    The epidemiology of variant Creutzfeldt-Jakob disease.
    Smith PG, Cousens SN, d’ Huillard Aignaux JN, Ward HJ, Will RG.
    Department of Infectious and Tropical Diseases, London School of Hygiene and Tropical Medicine, Keppel Street, London WC1E 7HT, UK. p.smith@lshtm.ac.uk
    Variant Creutzfeldt-Jakob disease (vCJD) was identified as a new disease in 1996. It was linked to infection with the bovine spongiform encephalopathy (BSE) agent although the epidemiological evidence for this was not strong, but later strain typing studies confirmed the association. The disease has affected predominantly young adults whose dietary and other characteristics are unexceptional compared to control groups, other than that all patients to date have been methoinine homozygous at codon 129 of the prion protein gene and the incidence has been about two times higher in the North of the UK. The number of cases in the 7 years after first identification of the disease has been considerably lower than initially feared, given the likely widespread exposure of the UK population to the BSE agent through contaminated beef products. Predictions of the possible future course of the epidemic have many associated uncertainties, but current mathematical models suggest that more than a few thousand cases is unlikely. Such modelling is limited by the absence of a test for infection with the vCJD agent. The development of a test that could be used on easily accessible tissue to detect infection early in the incubation period would not only advance understanding of the epidemiology of infection with the agent but would also aid the implementation of control measures to prevent potential iatrogenic spread.
    sease epidemics of at most several thousand cases.
    8. 2006 Jan;59(1):111-20.
    Risk factors for variant Creutzfeldt-Jakob disease: a case-control study.
    Rizikové faktory pro nemoc vCJD: případ-kontrolní studie.
    Ward HJ, Everington D, Cousens SN, Smith-Bathgate B, Leitch M, Cooper S, Heath C, Knight RS, Smith PG, Will RG.
    National Creutzfeldt-Jakob Disease Surveillance Unit, University of Edinburgh, Western General Hospital, Crewe Road, Edinburgh EH4 2XU, UK. h.ward@ed.ac.uk
    OBJECTIVE: To investigate the potential risk factors for variant Creutzfeldt-Jakob disease (VCJD) in the United Kingdom. METHODS: Definite and probable vCJD cases (n = 136) were residing in Great Britain at disease onset, and were referred between May 1995 and November 2003. Control subjects (n = 922) were recruited between 2002 and 2003, from 100 randomly selected geographical clusters sampled to represent the geographical distribution of vCJD.
    RESULTS: Reported frequent consumption of beef and beef products thought likely to contain mechanically recovered or head meat, or both, including burgers and meat pies, was associated with increased risk for vCJD, as was reported frequent chicken consumption. Surgical operations were generally similarly reported for cases and control subjects, with the exception of a small group of minor operations, possibly attributable to underreporting in control subjects. Cases and control subjects had similar reported occupational histories and exposure to animals. INTERPRETATION: These findings are consistent with dietary exposure to contaminated beef products being the main route of infection of vCJD, but recall bias cannot be excluded. There was no convincing evidence of increased risk through medical, surgical, or occupational exposure or exposure to animals.
    Sincerely
    Josef Hlasny, DVM,PhD., veterinary surgeon, Czech Republic

  • The UKBSEnvCJD only theory is bogus. has been since day one. when you have farmers and their wives dying from sporadic CJD, and these same farmers and their wives having herds of cattle infected with BSE, THEN SOMETHING IS TERRIBLY WRONG. sporadic CJD figures have been rising since BSE. just look at the figures. sporadic CJD means ROUTE AND SOURCE UNKNOWN, and there could be many right here in the USA from all the animal TSE documented. The USA has the most documented TSE in species than any other country, and that’s not counting the dogs and cats (yes i believe canine can contract a TSE as well), all these animals rendered and fed back to livestock producing animals and humans for years. c-BSE, l-BSE, and h-BSE all documented in North America. The USA has typical scrapie’s, and now the atypical Nor-98 scrapie documented, two strains of CWD in deer and elk, TME in mink, all this rendered and fed to humans and animals. sporadic CJD rising ???
    YOU are also wrong about no convincing evidence of medical, surgical, and or occupational exposure, I don’t care how many phd’s you have.
    Tuesday, August 12, 2008
    Biosafety in Microbiological and Biomedical Laboratories Fifth Edition 2007 (occupational exposure to prion diseases)
    http://creutzfeldt-jakob-disease.blogspot.com/2008/08/biosafety-in-microbiological-and.html
    Sunday, August 09, 2009
    CJD…Straight talk with…James Ironside…and…Terry Singeltary… 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/08/cjdstraight-talk-withjames.html
    Tuesday, August 18,
    2009 BSE-The Untold Story – joe gibbs and singeltary 1999 – 2009
    http://madcowusda.blogspot.com/2009/08/bse-untold-story-joe-gibbs-and.html
    Saturday, December 12, 2009
    103RD MEETING OF THE SPONGIFORM ENCEPHALOPATHY ADVISORY COMMITTEE
    http://seac992007.blogspot.com/2009/12/103rd-meeting-of-spongiform.html
    1: J Neurol Neurosurg Psychiatry 1994 Jun;57(6):757-8
    Transmission of Creutzfeldt-Jakob disease to a chimpanzee by electrodes contaminated during neurosurgery.
    Gibbs CJ Jr, Asher DM, Kobrine A, Amyx HL, Sulima MP, Gajdusek DC.
    Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892.
    Stereotactic multicontact electrodes used to probe the cerebral cortex of a middle aged woman with progressive dementia were previously implicated in the accidental transmission of Creutzfeldt-Jakob disease (CJD) to two younger patients. The diagnoses of CJD have been confirmed for all three cases. More than two years after their last use in humans, after three cleanings and repeated sterilisation in ethanol and formaldehyde vapour, the electrodes were implanted in the cortex of a chimpanzee. Eighteen months later the animal became ill with CJD. This finding serves to re-emphasise the potential danger posed by reuse of instruments contaminated with the agents of spongiform encephalopathies, even after scrupulous attempts to clean them.
    http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=8006664&dopt=Abstract
    Sunday, January 17, 2010
    Human tissue, recovered from a donor history indicated increased risk factors for Creutzfeldt-Jacob disease Lions Eye Bank
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/human-tissue-recovered-from-donor.html
    Saturday, January 16, 2010 Evidence For CJD TSE Transmission Via Endoscopes 1-24-3 re-Singeltary to Bramble et al
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/evidence-for-cjd-tse-transmission-via.html
    Friday, November 20, 2009
    SaBTO Advisory Committee on the Safety of Blood, Tissues and Organs Summary of the Eighth Meeting, 27 October 2009
    http://vcjdtransfusion.blogspot.com/2009/11/sabto-advisory-committee-on-safety-of.html
    Sunday, May 10, 2009
    Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)
    http://tseac.blogspot.com/2009/05/meeting-of-transmissible-spongiform.html
    Sunday, January 17, 2010
    Human tissue, recovered from a donor history indicated increased risk factors for Creutzfeldt-Jacob disease Lions Eye Bank
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/human-tissue-recovered-from-donor.html
    http://vcjdtransfusion.blogspot.com/2009/04/more-blood-products-collected-from.html
    Monday, August 17, 2009
    Transmissible Spongiform Encephalopathy Agents: Safe Working and the Prevention of Infection: Annex J,K, AND D Published: 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/08/transmissible-spongiform-encephalopathy.html
    Friday, July 17, 2009
    Revision to pre-surgical assessment of risk for vCJD in neurosurgery and eye surgery units Volume 3 No 28; 17 July 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/07/revision-to-pre-surgical-assessment-of.html
    Sunday, August 10, 2008
    A New Prionopathy OR more of the same old BSe and sporadic CJD
    http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html
    Monday, May 19, 2008
    SPORADIC CJD IN FARMERS, FARMERS WIVES, FROM FARMS WITH BSE HERD AND ABATTOIRS
    http://bseinquiry.blogspot.com/
    3. Neither Dr Will nor the CJD surveillance unit intend to disclose the existence of this case or make any comment at present unless it attracts media attention.
    snip…
    HUMAN CASE DETAILS CONFIDENTIAL
    snip…
    6. CJD IN FARMERS
    The second annual report on CJD surveillance in the UK, which is about to be published, gives occupational history details of 29 definite and probable CJD cases recorded in people who had a history of employment at any time in particular occupational groups of potential significance for the occurrence of the disease. The 29 cases were amongst 95 diagnosed over a 3 year period: the other 66 cases did not fall into such occupational groups.
    These relevant details are:-
    MEDICAL/PARAMEDICAL/DENTISTRY 7
    ANIMAL LABORATORY 1
    PHARMACEUTICAL LABORATORY 0
    RESEARCH LABORATORY 0
    FARMERS/VETERINARY SURGEONS 7
    BUTCHERS/ABATTOIR WORKERS/OCCUPATION INVOLVING DIRECT CONTACT WITH ANIMAL OR CARCASES 5
    OCCUPATION INVOLVING ANIMAL PRODUCTS 9
    snip… full text ;
    http://www.bseinquiry.gov.uk/files/yb/1993/07/19001001.pdf
    2. snip… Over a 5 year period, which is the time period on which the advice from Professor Smith and Dr. Gore was based, and assuming a population of 120,000 dairy farm workers, and an annual incidence of 1 per million cases of CJD in the general population, a DAIRY FARM WORKER IS 5 TIMES MORE LIKELY THAN an individual in the general population to develop CJD. Using the actual current annual incidence of CJD in the UK of 0.7 per million, this figure becomes 7.5 TIMES.
    3. You will recall that the advice provided by Professor Smith in 1993 and by Dr. Gore this month used the sub-population of dairy farm workers who had had a case of BSE on their farms – 63,000, which is approximately half the number of dairy farm workers – as a denominator. If the above sums are repeated using this denominator population, taking an annual incidence in the general population of 1 per million the observed rate in this sub-population is 10 TIMES, and taking an annual incidence of 0.7 per million, IT IS 15 TIMES (THE ”WORST CASE” SCENARIO) than that in the general population…
    http://www.bseinquiry.gov.uk/files/yb/1995/01/31004001.pdf
    CHANGING SCIENCE TO FIT YOUR INDUSTRY NEEDS COVER-UP IN FULL MODE NOW
    PROBLEM
    7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x).
    IP PS(L) wishes to probe this further we think it best to explain the matter VERBALLY. The problem is how to present the findings in this year’s annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stores. (or the facts…TSS)
    http://www.bseinquiry.gov.uk/files/yb/1994/07/00001001.pdf
    A REVISED VERSION WHERE THE FOLLOWING WAS MADE TO BE REMOVED FROM SCIENTIFIC FINDINGS. …TSS
    ”This year’s findings show a number of associations but the strongest is for veal.”
    A BIG LINE WAS DRAWN THROUGH THAT SENTENCE TO BE REMOVED DUE TO THE FOLLOWING. THIS IS THE NEXT SENTENCE ;
    ”This is of considerable concern given recent developments. In particular, Ministers will be particularly concerned about the European dimension given the recent troubles with the Germans.”
    YOU can see the beginning of the ukbsenvCJD only theory beginning to unfold now. full text of this ukbsenvcjd only conspiracy can be seen here. …TSS
    POLICY RESTRICTED
    http://www.bseinquiry.gov.uk/files/yb/1994/07/00001001.pdf
    see full text sporadic CJD the big lie;
    CJD IN AN INDIVIDUAL OCCUPATIONALLY EXPOSED TO BSE
    ii. on page 2 the sentence ”He had drunk pooled milk from the herd which included that from the affected animal” will mislead the uninformed. It needs to be made clear that milk from a cow which is suspected to be affected with BSE cannot be drunk or added to the bulk milk produced by the rest of the herd.
    iii. in the final paragraph I suggest that the phrase ”and a causal link with BSE is at most conjectural” BE DELETED: the first paragraph of the sentence would then stand as a clear statement that the CJD case was likely to have been a CHANCE PHENOMENON.
    http://www.bseinquiry.gov.uk/files/yb/1993/02/15003001.pdf
    ”DH is aware of a second case of CJD in a dairy farmer who has had BSE in his herd. We cannot comment on the details of the case, but we know of nothing to suggest this is anthing other than a sporadic case of CJD. ………
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12001001.pdf
    IF PRESSED:
    The numbers concerned are very small, and it is not possible to draw any conclusions from such small numbers. This issue is being considered by the Government’s expert advisers….
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12002001.pdf
    THE FARMER IS THOUGHT TO HAVE HAD AT LEAST TWO CASES OF BSE IN HIS HERD, which were diagnosed in 1992. The farmer is reported to have asssisted in calving and to have drunk milk from his herd. This does not suggest that this is anything other than a sporadic case of CJD. …
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12003001.pdf
    CONFIDENTIAL
    CONFIRMED CASE OF CJD IN DAIRY FARMER
    http://www.bseinquiry.gov.uk/files/yb/1993/07/14003001.pdf
    3. Neither Dr Will nor the CJD surveillance unit intend to disclose the existence of this case or make any comment at present unless it attracts media attention.
    snip…
    HUMAN CASE DETAILS CONFIDENTIAL
    snip…
    http://cjdmadcowbaseoct2007.blogspot.com/2008/07/new-prionopathy-update-july-10-2008.html
    Hound Survey
    http://web.archive.org/web/20010305222642/www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf
    2005 DEFRA Department for Environment, Food & Rural Affairs
    Area 307, London, SW1P 4PQ Telephone: 0207 904 6000 Direct line: 0207 904 6287 E-mail: h.mcdonagh.defra.gsi.gov.uk
    GTN: FAX:
    Mr T S Singeltary P.O. Box 42 Bacliff Texas USA 77518
    21 November 2001
    Dear Mr Singeltary
    TSE IN HOUNDS
    Thank you for e-mail regarding the hounds survey. I am sorry for the long delay in responding.
    As you note, the hound survey remains unpublished. However the Spongiform Encephalopathy Advisory Committee (SEAC), the UK Government’s independent Advisory Committee on all aspects related to BSE-like disease, gave the hound study detailed consideration at their meeting in January 1994. As a summary of this meeting published in the BSE inquiry noted, the Committee were clearly concerned about the work that had been carried out, concluding that there had clearly been problems with it, particularly the control on the histology, and that it was more or less inconclusive. However was agreed that there should be a re-evaluation of the pathological material in the study.
    Later, at their meeting in June 95, The Committee re-evaluated the hound study to see if any useful results could be gained from it. The Chairman concluded that there were varying opinions within the Committee on further work. It did not suggest any further transmission studies and thought that the lack of clinical data was a major weakness.
    Overall, it is clear that SEAC had major concerns about the survey as conducted. As a result it is likely that the authors felt that it would not stand up to r~eer review and hence it was never published. As noted above, and in the detailed minutes of the SEAC meeting in June 95, SEAC considered whether additional work should be performed to examine dogs for evidence of TSE infection. Although the Committee had mixed views about the merits of conducting further work, the Chairman noted that when the Southwood Committee made their recommendation to complete an assessment of possible spongiform disease in dogs, no TSEs had been identified in other species and hence dogs were perceived as a high risk population and worthy of study. However subsequent to the original recommendation, made in 1990, a number of other species had been identified with TSE ( e.g. cats) so a study in hounds was less
    critical. For more details see- http://www.bseinquiry.gov.uk/files/yb/1995/06/21005001.pdf
    As this study remains unpublished, my understanding is that the ownership of the data essentially remains with the original researchers. Thus unfortunately, I am unable to help with your request to supply information on the hound survey directly. My only suggestion is that you contact one of the researchers originally involved in the project, such as Gerald Wells. He can be contacted at the following address.
    Dr Gerald Wells, Veterinary Laboratories Agency, New Haw, Addlestone, Surrey, KT 15 3NB, UK
    You may also wish to be aware that since November 1994 all suspected cases of spongiform encephalopathy in animals and poultry were made notifiable. Hence since that date there has been a requirement for vets to report any suspect SE in dogs for further investigation. To date there has never been positive identification of a TSE in a dog.
    I hope this is helpful
    Yours sincerely 4
    HUGH MCDONAGH BSE CORRESPONDENCE SECTION
    IN CONFIDENCE
    CONCEPT NOT FOR FURTHER STUDY OF MATERIAL OBTAINED IN A SURVEY OF HOUNDS FOR EVIDENCE OF A SCRAPIE-LIKE SPONGIFORM ENCEPHALOPATHY (SE)
    snip…
    b) Fibrillar material closely similar to SAF, found in BSE/Scrapie, was observed in 19 (4.3%) cases, all of which were hounds > 7 years of age. 14/19 of these suspected SAF results correlated with cases in the unresolveable histopathological category.
    snip…
    The following proposals address the hypothesis that the hound survey observations represent a PrP related or scrapie-like disease of dogs in which the pathological response, and possible the spread of infectivity, is neuroanatomically localized. By inference this could also mean that the disorder is clinically silent and non-progressive.
    http://www.bseinquiry.gov.uk/files/yb/1995/02/09001001.pdf
    http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html
    http://www.humanitarian.net/law/biodefense/bse_12004.html
    http://www.mad-cow.org/00/aug00_late_news.html#ggg
    In Confidence – Perceptions of unconventional slow virus diseases of animals in the USA – APRIL-MAY 1989 – G A H Wells
    http://www.bseinquiry.gov.uk/files/mb/m11b/tab01.pdf
    2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006
    http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html
    I ask Professor Kong ;
    Thursday, December 04, 2008 3:37 PM Subject: RE: re–Chronic Wating Disease (CWD) and Bovine Spongiform Encephalopathies (BSE): Public Health Risk Assessment
    ”IS the h-BSE more virulent than typical BSE as well, or the same as cBSE, or less virulent than cBSE? just curious…..”
    Professor Kong reply ;
    …..snip
    ”As to the H-BSE, we do not have sufficient data to say one way or another, but we have found that H-BSE can infect humans. I hope we could publish these data once the study is complete.
    Thanks for your interest.”
    Best regards,
    Qingzhong Kong, PhD Associate Professor Department of Pathology Case Western Reserve University Cleveland, OH 44106 USA
    END…TSS
    P02.35
    Molecular Features of the Protease-resistant Prion Protein (PrPres) in H-type BSE
    Biacabe, A-G1; Jacobs, JG2; Gavier-Widén, D3; Vulin, J1; Langeveld, JPM2; Baron, TGM1 1AFSSA, France; 2CIDC-Lelystad, Netherlands; 3SVA, Sweden
    Western blot analyses of PrPres accumulating in the brain of BSE-infected cattle have demonstrated 3 different molecular phenotypes regarding to the apparent molecular masses and glycoform ratios of PrPres bands. We initially described isolates (H-type BSE) essentially characterized by higher PrPres molecular mass and decreased levels of the diglycosylated PrPres band, in contrast to the classical type of BSE. This type is also distinct from another BSE phenotype named L-type BSE, or also BASE (for Bovine Amyloid Spongiform Encephalopathy), mainly characterized by a low representation of the diglycosylated PrPres band as well as a lower PrPres molecular mass. Retrospective molecular studies in France of all available BSE cases older than 8 years old and of part of the other cases identified since the beginning of the exhaustive surveillance of the disease in 20001 allowed to identify 7 H-type BSE cases, among 594 BSE cases that could be classified as classical, L- or H-type BSE. By Western blot analysis of H-type PrPres, we described a remarkable specific feature with antibodies raised against the C-terminal region of PrP that demonstrated the existence of a more C-terminal cleaved form of PrPres (named PrPres#2 ), in addition to the usual PrPres form (PrPres #1). In the unglycosylated form, PrPres #2 migrates at about 14 kDa, compared to 20 kDa for PrPres #1. The proportion of the PrPres#2 in cattle seems to by higher compared to the PrPres#1. Furthermore another PK–resistant fragment at about 7 kDa was detected by some more N-terminal antibodies and presumed to be the result of cleavages of both N- and C-terminal parts of PrP. These singular features were maintained after transmission of the disease to C57Bl/6 mice. The identification of these two additional PrPres fragments (PrPres #2 and 7kDa band) reminds features reported respectively in sporadic Creutzfeldt-Jakob disease and in Gerstmann-Sträussler-Scheinker (GSS) syndrome in humans.
    http://www.neuroprion.com/pdf_docs/conferences/prion2007/abstract_book.pdf
    Research Project: Study of Atypical Bse Location: Virus and Prion Diseases of Livestock
    Project Number: 3625-32000-086-05 Project Type: Specific Cooperative Agreement
    Start Date: Sep 15, 2004 End Date: Sep 14, 2009
    Objective: The objective of this cooperative research project with Dr. Maria Caramelli from the Italian BSE Reference Laboratory in Turin, Italy, is to conduct comparative studies with the U.S. bovine spongiform encephalopathy (BSE) isolate and the atypical BSE isolates identified in Italy. The studies will cover the following areas: 1. Evaluation of present diagnostics tools used in the U.S. for the detection of atypical BSE cases. 2. Molecular comparison of the U.S. BSE isolate and other typical BSE isolates with atypical BSE cases. 3. Studies on transmissibility and tissue distribution of atypical BSE isolates in cattle and other species.
    Approach: This project will be done as a Specific Cooperative Agreement with the Italian BSE Reference Laboratory, Istituto Zooprofilattico Sperimentale del Piemonte, in Turin, Italy. It is essential for the U.S. BSE surveillance program to analyze the effectiveness of the U.S diagnostic tools for detection of atypical cases of BSE. Molecular comparisons of the U.S. BSE isolate with atypical BSE isolates will provide further characterization of the U.S. BSE isolate. Transmission studies are already underway using brain homogenates from atypical BSE cases into mice, cattle and sheep. It will be critical to see whether the atypical BSE isolates behave similarly to typical BSE isolates in terms of transmissibility and disease pathogenesis. If transmission occurs, tissue distribution comparisons will be made between cattle infected with the atypical BSE isolate and the U.S. BSE isolate. Differences in tissue distribution could require new regulations regarding specific risk material (SRM) removal.
    http://www.ars.usda.gov/research/projects/projects.htm?ACCN_NO=408490
    Wednesday, February 11, 2009
    Atypical BSE North America Update February 2009
    Both of the BSE cases ascertained in the US native-born cattle were atypical cases (H-type), which contributed to the initial ambiguity of the diagnosis. 174, 185 In Canada, there have been 2 atypical BSE cases in addition to the 14 cases of the classic UK strain of BSE2: one was the H-type, and the other was of the L-type.198
    snip…end
    source :
    Enhanced Abstract Journal of the American Veterinary Medical Association January 1, 2009, Vol. 234, No. 1, Pages 59-72
    Bovine spongiform encephalopathy
    Jane L. Harman, DVM, PhD; Christopher J. Silva, PhD
    http://avmajournals.avma.org/doi/ref/10.2460/javma.234.1.59
    Atypical BSE North America Update February 2009
    http://bse-atypical.blogspot.com/2009/02/atypical-bse-north-america-update.html
    snip…
    http://bse-atypical.blogspot.com/2009/04/transmission-of-atypical-bovine.html
    Monday, October 19, 2009
    Atypical BSE, BSE, and other human and animal TSE in North America Update October 19, 2009
    http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html
    Thursday, January 07, 2010
    Scrapie and Nor-98 Scrapie November 2009 Monthly Report Fiscal Year 2010 and FISCAL YEAR 2008
    http://scrapie-usa.blogspot.com/2010/01/scrapie-and-nor-98-scrapie-november.html
    CJD USA RISING, with UNKNOWN PHENOTYPE ;
    5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.
    http://www.cjdsurveillance.com/pdf/case-table.pdf
    Saturday, January 2, 2010
    Human Prion Diseases in the United States January 1, 2010 ***FINAL***
    http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html
    my comments to PLosone here ;
    http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd
    Terry S. Singeltary Sr.
    P.O. Box 42
    Bacliff, Texas USA 77518

  • Hello Dr. Hlasny, DVM, PhD, veterinary surgeon Czech Republic.
    I kindly disagree with your assessment for the following reasons.
    There is much more to this nightmare than the UKBSEnvCJD hamburger eating adolescents only theory. WE are missing the bigger picture, i.e. 85%+ of all the rest of the cases i.e. sporadic Creutzfeldt Jakob Disease, and all it’s phenotypes, and they are mounting. please remember ;
    BSE prions propagate as either variant CJD-like or sporadic CJD-like prion strains in transgenic mice expressing human prion protein
    Emmanuel A. Asante, Jacqueline M. Linehan, Melanie Desbruslais, Susan Joiner, Ian Gowland, Andrew L. Wood, Julie Welch, Andrew F. Hill, Sarah E. Lloyd, Jonathan D.F. Wadsworth, and John Collinge1 MRC Prion Unit and Department of Neurodegenerative Disease, Institute of Neurology, University College, Queen Square, London WC1N 3BG, UK 1Corresponding author e-mail: j.collinge@prion.ucl.ac.ukReceived August 1, 2002; Revised September 24, 2002; Accepted October 17, 2002.
    http://www.ncbi.nlm.nih.gov/pmc/articles/PMC136957/?tool=pubmed
    Monday, May 19, 2008
    SPORADIC CJD IN FARMERS, FARMERS WIVES, FROM FARMS WITH BSE HERD AND ABATTOIRS
    http://bseinquiry.blogspot.com/
    3. Neither Dr Will nor the CJD surveillance unit intend to disclose the existence of this case or make any comment at present unless it attracts media attention.
    snip…
    HUMAN CASE DETAILS CONFIDENTIAL
    snip…
    6. CJD IN FARMERS
    The second annual report on CJD surveillance in the UK, which is about to be published, gives occupational history details of 29 definite and probable CJD cases recorded in people who had a history of employment at any time in particular occupational groups of potential significance for the occurrence of the disease. The 29 cases were amongst 95 diagnosed over a 3 year period: the other 66 cases did not fall into such occupational groups.
    These relevant details are:-
    MEDICAL/PARAMEDICAL/DENTISTRY 7
    ANIMAL LABORATORY 1
    PHARMACEUTICAL LABORATORY 0
    RESEARCH LABORATORY 0
    FARMERS/VETERINARY SURGEONS 7
    BUTCHERS/ABATTOIR WORKERS/OCCUPATION INVOLVING DIRECT CONTACT WITH ANIMAL OR CARCASES 5
    OCCUPATION INVOLVING ANIMAL PRODUCTS 9
    snip… full text ;
    http://www.bseinquiry.gov.uk/files/yb/1993/07/19001001.pdf
    2. snip…
    Over a 5 year period, which is the time period on which the advice from Professor Smith and Dr. Gore was based, and assuming a population of 120,000 dairy farm workers, and an annual incidence of 1 per million cases of CJD in the general population, a DAIRY FARM WORKER IS 5 TIMES MORE LIKELY THAN an individual in the general population to develop CJD. Using the actual current annual incidence of CJD in the UK of 0.7 per million, this figure becomes 7.5 TIMES.
    3. You will recall that the advice provided by Professor Smith in 1993 and by Dr. Gore this month used the sub-population of dairy farm workers who had had a case of BSE on their farms – 63,000, which is approximately half the number of dairy farm workers – as a denominator. If the above sums are repeated using this denominator population, taking an annual incidence in the general population of 1 per million the observed rate in this sub-population is 10 TIMES, and taking an annual incidence of 0.7 per million, IT IS 15 TIMES (THE ”WORST CASE” SCENARIO) than that in the general population…
    http://www.bseinquiry.gov.uk/files/yb/1995/01/31004001.pdf
    CHANGING SCIENCE TO FIT YOUR INDUSTRY NEEDS COVER-UP IN FULL MODE NOW
    PROBLEM
    7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x).
    IP PS(L) wishes to probe this further we think it best to explain the matter VERBALLY. The problem is how to present the findings in this year’s annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stores. (or the facts…TSS)
    http://www.bseinquiry.gov.uk/files/yb/1994/07/00001001.pdf
    VEAL AND BSE
    A REVISED VERSION WHERE THE FOLLOWING WAS MADE TO BE REMOVED FROM SCIENTIFIC FINDINGS. …TSS
    ”This year’s findings show a number of associations but the strongest is for veal.”
    A BIG LINE WAS DRAWN THROUGH THAT SENTENCE TO BE REMOVED DUE TO THE FOLLOWING. THIS IS THE NEXT SENTENCE ;
    ”This is of considerable concern given recent developments. In particular, Ministers will be particularly concerned about the European dimension given the recent troubles with the Germans.”
    YOU can see the beginning of the ukbsenvCJD only theory beginning to unfold now. full text of this ukbsenvcjd only conspiracy can be seen here. …TSS
    POLICY RESTRICTED
    http://www.bseinquiry.gov.uk/files/yb/1994/07/00001001.pdf
    see full text sporadic CJD the big lie;
    CJD IN AN INDIVIDUAL OCCUPATIONALLY EXPOSED TO BSE
    ii. on page 2 the sentence ”He had drunk pooled milk from the herd which included that from the affected animal” will mislead the uninformed. It needs to be made clear that milk from a cow which is suspected to be affected with BSE cannot be drunk or added to the bulk milk produced by the rest of the herd.
    iii. in the final paragraph I suggest that the phrase ”and a causal link with BSE is at most conjectural” BE DELETED: the first paragraph of the sentence would then stand as a clear statement that the CJD case was likely to have been a CHANCE PHENOMENON.
    http://www.bseinquiry.gov.uk/files/yb/1993/02/15003001.pdf
    ”DH is aware of a second case of CJD in a dairy farmer who has had BSE in his herd. We cannot comment on the details of the case, but we know of nothing to suggest this is anthing other than a sporadic case of CJD. ………
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12001001.pdf
    IF PRESSED:
    The numbers concerned are very small, and it is not possible to draw any conclusions from such small numbers. This issue is being considered by the Government’s expert advisers….
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12002001.pdf
    THE FARMER IS THOUGHT TO HAVE HAD AT LEAST TWO CASES OF BSE IN HIS HERD, which were diagnosed in 1992. The farmer is reported to have asssisted in calving and to have drunk milk from his herd. This does not suggest that this is anything other than a sporadic case of CJD. …
    http://www.bseinquiry.gov.uk/files/yb/1993/07/12003001.pdf
    CONFIDENTIAL
    CONFIRMED CASE OF CJD IN DAIRY FARMER
    http://www.bseinquiry.gov.uk/files/yb/1993/07/14003001.pdf
    2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006
    http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html
    I ask Professor Kong ;
    Thursday, December 04, 2008 3:37 PM Subject: RE: re–Chronic Wating Disease (CWD) and Bovine Spongiform Encephalopathies (BSE): Public Health Risk Assessment
    ”IS the h-BSE more virulent than typical BSE as well, or the same as cBSE, or less virulent than cBSE? just curious…..”
    Professor Kong reply ;
    …..snip
    ”As to the H-BSE, we do not have sufficient data to say one way or another, but we have found that H-BSE can infect humans. I hope we could publish these data once the study is complete.
    Thanks for your interest.”
    Best regards,
    Qingzhong Kong, PhD Associate Professor Department of Pathology Case Western Reserve University Cleveland, OH 44106 USA
    END…TSS
    P02.35
    Molecular Features of the Protease-resistant Prion Protein (PrPres) in H-type BSE
    Biacabe, A-G1; Jacobs, JG2; Gavier-Widén, D3; Vulin, J1; Langeveld, JPM2; Baron, TGM1 1AFSSA, France; 2CIDC-Lelystad, Netherlands; 3SVA, Sweden
    Western blot analyses of PrPres accumulating in the brain of BSE-infected cattle have demonstrated 3 different molecular phenotypes regarding to the apparent molecular masses and glycoform ratios of PrPres bands. We initially described isolates (H-type BSE) essentially characterized by higher PrPres molecular mass and decreased levels of the diglycosylated PrPres band, in contrast to the classical type of BSE. This type is also distinct from another BSE phenotype named L-type BSE, or also BASE (for Bovine Amyloid Spongiform Encephalopathy), mainly characterized by a low representation of the diglycosylated PrPres band as well as a lower PrPres molecular mass. Retrospective molecular studies in France of all available BSE cases older than 8 years old and of part of the other cases identified since the beginning of the exhaustive surveillance of the disease in 20001 allowed to identify 7 H-type BSE cases, among 594 BSE cases that could be classified as classical, L- or H-type BSE. By Western blot analysis of H-type PrPres, we described a remarkable specific feature with antibodies raised against the C-terminal region of PrP that demonstrated the existence of a more C-terminal cleaved form of PrPres (named PrPres#2 ), in addition to the usual PrPres form (PrPres #1). In the unglycosylated form, PrPres #2 migrates at about 14 kDa, compared to 20 kDa for PrPres #1. The proportion of the PrPres#2 in cattle seems to by higher compared to the PrPres#1. Furthermore another PK–resistant fragment at about 7 kDa was detected by some more N-terminal antibodies and presumed to be the result of cleavages of both N- and C-terminal parts of PrP. These singular features were maintained after transmission of the disease to C57Bl/6 mice. The identification of these two additional PrPres fragments (PrPres #2 and 7kDa band) reminds features reported respectively in sporadic Creutzfeldt-Jakob disease and in Gerstmann-Sträussler-Scheinker (GSS) syndrome in humans.
    http://www.neuroprion.com/pdf_docs/conferences/prion2007/abstract_book.pdf
    Research Project: Study of Atypical Bse Location: Virus and Prion Diseases of Livestock
    Project Number: 3625-32000-086-05 Project Type: Specific Cooperative Agreement
    Start Date: Sep 15, 2004 End Date: Sep 14, 2009
    Objective: The objective of this cooperative research project with Dr. Maria Caramelli from the Italian BSE Reference Laboratory in Turin, Italy, is to conduct comparative studies with the U.S. bovine spongiform encephalopathy (BSE) isolate and the atypical BSE isolates identified in Italy. The studies will cover the following areas: 1. Evaluation of present diagnostics tools used in the U.S. for the detection of atypical BSE cases. 2. Molecular comparison of the U.S. BSE isolate and other typical BSE isolates with atypical BSE cases. 3. Studies on transmissibility and tissue distribution of atypical BSE isolates in cattle and other species.
    Approach: This project will be done as a Specific Cooperative Agreement with the Italian BSE Reference Laboratory, Istituto Zooprofilattico Sperimentale del Piemonte, in Turin, Italy. It is essential for the U.S. BSE surveillance program to analyze the effectiveness of the U.S diagnostic tools for detection of atypical cases of BSE. Molecular comparisons of the U.S. BSE isolate with atypical BSE isolates will provide further characterization of the U.S. BSE isolate. Transmission studies are already underway using brain homogenates from atypical BSE cases into mice, cattle and sheep. It will be critical to see whether the atypical BSE isolates behave similarly to typical BSE isolates in terms of transmissibility and disease pathogenesis. If transmission occurs, tissue distribution comparisons will be made between cattle infected with the atypical BSE isolate and the U.S. BSE isolate. Differences in tissue distribution could require new regulations regarding specific risk material (SRM) removal.
    http://www.ars.usda.gov/research/projects/projects.htm?ACCN_NO=408490
    Wednesday, February 11, 2009
    Atypical BSE North America Update February 2009
    Both of the BSE cases ascertained in the US native-born cattle were atypical cases (H-type), which contributed to the initial ambiguity of the diagnosis. 174, 185 In Canada, there have been 2 atypical BSE cases in addition to the 14 cases of the classic UK strain of BSE2: one was the H-type, and the other was of the L-type.198
    snip…end
    source :
    Enhanced Abstract Journal of the American Veterinary Medical Association January 1, 2009, Vol. 234, No. 1, Pages 59-72
    Bovine spongiform encephalopathy
    Jane L. Harman, DVM, PhD; Christopher J. Silva, PhD
    http://avmajournals.avma.org/doi/ref/10.2460/javma.234.1.59
    Atypical BSE North America Update February 2009
    http://bse-atypical.blogspot.com/2009/02/atypical-bse-north-america-update.html
    snip…
    http://bse-atypical.blogspot.com/2009/04/transmission-of-atypical-bovine.html
    Monday, October 19, 2009
    Atypical BSE, BSE, and other human and animal TSE in North America Update October 19, 2009
    http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html
    Tuesday, August 12, 2008
    Biosafety in Microbiological and Biomedical Laboratories Fifth Edition 2007 (occupational exposure to prion diseases)
    http://creutzfeldt-jakob-disease.blogspot.com/2008/08/biosafety-in-microbiological-and.html
    Sunday, August 09, 2009
    CJD…Straight talk with…James Ironside…and…Terry Singeltary… 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/08/cjdstraight-talk-withjames.html
    Tuesday, August 18,
    2009 BSE-The Untold Story – joe gibbs and singeltary 1999 – 2009
    http://madcowusda.blogspot.com/2009/08/bse-untold-story-joe-gibbs-and.html
    : J Neurol Neurosurg Psychiatry 1994 Jun;57(6):757-8
    Transmission of Creutzfeldt-Jakob disease to a chimpanzee by electrodes contaminated during neurosurgery.
    Gibbs CJ Jr, Asher DM, Kobrine A, Amyx HL, Sulima MP, Gajdusek DC.
    Laboratory of Central Nervous System Studies, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892.
    Stereotactic multicontact electrodes used to probe the cerebral cortex of a middle aged woman with progressive dementia were previously implicated in the accidental transmission of Creutzfeldt-Jakob disease (CJD) to two younger patients. The diagnoses of CJD have been confirmed for all three cases. More than two years after their last use in humans, after three cleanings and repeated sterilisation in ethanol and formaldehyde vapour, the electrodes were implanted in the cortex of a chimpanzee. Eighteen months later the animal became ill with CJD. This finding serves to re-emphasise the potential danger posed by reuse of instruments contaminated with the agents of spongiform encephalopathies, even after scrupulous attempts to clean them.
    http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=8006664&dopt=Abstract
    Sunday, January 17, 2010
    Human tissue, recovered from a donor history indicated increased risk factors for Creutzfeldt-Jacob disease Lions Eye Bank
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/human-tissue-recovered-from-donor.html
    Saturday, January 16, 2010
    Evidence For CJD TSE Transmission Via Endoscopes 1-24-3 re-Singeltary to Bramble et al
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/evidence-for-cjd-tse-transmission-via.html
    Friday, November 20, 2009
    SaBTO Advisory Committee on the Safety of Blood, Tissues and Organs Summary of the Eighth Meeting, 27 October 2009
    http://vcjdtransfusion.blogspot.com/2009/11/sabto-advisory-committee-on-safety-of.html
    Sunday, May 10, 2009
    Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)
    http://tseac.blogspot.com/2009/05/meeting-of-transmissible-spongiform.html
    Sunday, January 17, 2010
    Human tissue, recovered from a donor history indicated increased risk factors for Creutzfeldt-Jacob disease Lions Eye Bank
    http://creutzfeldt-jakob-disease.blogspot.com/2010/01/human-tissue-recovered-from-donor.html
    http://vcjdtransfusion.blogspot.com/2009/04/more-blood-products-collected-from.html
    Monday, August 17, 2009
    Transmissible Spongiform Encephalopathy Agents: Safe Working and the Prevention of Infection: Annex J,K, AND D Published: 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/08/transmissible-spongiform-encephalopathy.html
    Friday, July 17, 2009
    Revision to pre-surgical assessment of risk for vCJD in neurosurgery and eye surgery units Volume 3 No 28; 17 July 2009
    http://creutzfeldt-jakob-disease.blogspot.com/2009/07/revision-to-pre-surgical-assessment-of.html
    Sunday, August 10, 2008
    A New Prionopathy OR more of the same old BSe and sporadic CJD
    http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html
    Monday, May 19, 2008
    SPORADIC CJD IN FARMERS, FARMERS WIVES, FROM FARMS WITH BSE HERD AND ABATTOIRS
    http://bseinquiry.blogspot.com/
    CJD USA RISING, with UNKNOWN PHENOTYPE ;
    5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.
    http://www.cjdsurveillance.com/pdf/case-table.pdf
    Saturday, January 2, 2010
    Human Prion Diseases in the United States January 1, 2010 ***FINAL***
    http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html
    my comments to PLosone here ;
    http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd
    Terry S. Singeltary Sr.
    P.O. Box 42
    Bacliff, Texas USA 77518